Thursday, 19 September 2013

20 September, 2013

Gene discovery could lead to new HIV treatments
Scientists have identified a new gene that may have the ability to prevent HIV from spreading after the virus enters the body.
The study led by King's College London is the first to identify a role for the human MX2 gene in inhibiting Human immunodeficiency virus (HIV), the virus that causes AIDS.
Researchers said this gene could be a new target for effective, less toxic treatments where the body's own natural defence system is mobilised against the virus.
Scientists carried out experiments on human cells in the lab, introducing the virus to two different cell lines and observing the effects. In one cell line the MX2 gene was expressed or 'switched on', and in the other it was not, or 'silenced'. They saw that in the cells where MX2 was silenced, the virus replicated and spread.
In the cells where the MX2 gene was expressed, the virus was not able to replicate and new viruses were not produced. The work was led by Dr Caroline Goujon and Professor Mike Malim at the Department of Infectious Diseases, King's College London.
"This is an extremely exciting finding which advances our understanding of how HIV virus interacts with the immune system and opens up opportunities to develop new therapies to treat the disease," Malim said. "Until now we knew very little about the MX2 gene, but now we recognise both its potent anti-viral function and a key point of vulnerability in the life cycle of HIV.
"Developing drugs to stimulate the body's natural inhibitors is a very important approach because you are triggering a natural process and therefore won't have the problem of drug resistance. "There are two possible routes - it may be possible to develop either a molecule that mimics the role of MX2 or a drug which activates the gene's natural capabilities.
"Although people with HIV are living longer, healthier lives with the virus thanks to current effective treatments, they can often be toxic for the body and drug resistance can become an issue with long-term use. "It is important to continue to find new ways of mobilising the body's natural defence systems and this gene appears to be a key player in establishing viral control in people with HIV," he said.
20.09.2013
Heart disease patients with positive attitudes live longer
Heart disease patients with positive attitudes are more likely to exercise and live longer, a new study has found. Researchers found that heart disease patients with a B positive attitude were 42 per cent less likely to die over a five-year period than those with a negative attitude.
Researchers used a questionnaire to assess the moods of 600 ischemic heart disease patients in a Denmark hospital. Ischemic heart disease, also called coronary artery disease, is caused by narrowed arteries that don't provide enough blood and oxygen to the heart.
After a five-year follow-up period, researchers found that the most positive patients exercised more and had a 42 per cent less chance of dying for any reason during the follow-up period; deaths were less than 10 per cent.
Among patients with less positive attitudes, 50 deaths occurred (16.5 per cent).
Positive mood and exercise also cut the risk of heart-related hospitalisations, the study found. Exercise levels the playing field between positive and negative patients, researchers said. So the differences in death rates between upbeat and sad heart patients weren't as striking when both groups exercised.
However, information on the types and amounts of exercise were not available.
"We should focus not only on increasing positive attitude in cardiac rehabilitation, but also make sure that patients perform exercise on a regular basis, as exercise is associated with both increased levels of optimism and better health," said Susanne S Pedersen, one of the study authors and professor of cardiac psychology, the Department of Medical and Clinical Psychology, Tilburg University, the Netherlands.
She is also adjunct professor of cardiac psychology, the University of Southern Denmark and Odense University Hospital, Denmark.
The study was published in the American Heart Association journal Circulation: Cardiovascular Quality and Outcomes.
20.09.2013








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